Wellcome

Wound healing biomaterials. Volume 1, Therapies and regeneration / edited by Magnus S. �Agren.

Contributor(s): �Agren, Magnus S [editor.]Material type: TextTextSeries: Woodhead Publishing series in biomaterials ; no. 114.Publisher: Duxford, UK : Woodhead Publishing is an imprint of Elsevier, 2016Description: 1 online resourceContent type: text Media type: computer Carrier type: online resourceISBN: 9780081006054; 0081006055Other title: Therapies and regenerationSubject(s): Wound healing | Biomedical materials | MEDICAL / Surgery / General | Biomedical materials | Wound healingGenre/Form: Electronic books.DDC classification: 617.1/4 LOC classification: RD94NLM classification: WO 185Online resources: ScienceDirect
Contents:
Front Cover; Wound Healing Biomaterials -- Volume 1; Related titles; Wound Healing Biomaterials: Volume 1: Therapies and Regeneration; Copyright; Contents; List of contributors; Woodhead Publishing Series in Biomaterials; One -- Fundamentals and strategies for wound healing; 1 -- Wound healing; 1.1 Introduction; 1.2 Skin layers; 1.3 Phases of wound healing; 1.3.1 Hemostasis and inflammation; 1.3.2 Proliferation; 1.3.3 Maturation and remodeling; 1.3.4 Wound contraction; 1.4 Growth factors and wound healing; 1.5 Acute and chronic wounds; 1.5.1 Acute wounds; 1.5.2 Chronic wounds.
1.5.2.1 Pressure ulcers1.5.2.2 Diabetic foot ulcers; 1.5.2.3 Arterial occlusive; 1.5.2.4 Venous stasis; 1.5.2.5 Lymphedema; 1.5.2.6 Calciphylaxis; 1.5.2.7 Warfarin-induced skin necrosis; 1.5.2.8 Infection; 1.6 Excessive scarring; 1.6.1 Hypertrophic scar; 1.6.2 Keloid; 1.6.3 Adhesions; 1.6.4 Pathophysiology; 1.6.5 Skin and scar evaluation; 1.6.6 Available treatment methods; 1.6.6.1 Intralesional injections; 1.6.6.2 Silicone gel; 1.6.6.3 Radiation; 1.6.6.4 Laser therapy; 1.6.6.5 Pressure therapy; 1.6.6.6 Surgery; 1.7 Burns; 1.7.1 Burn severity; 1.7.1.1 Superficial burns.
1.7.1.2 Superficial partial-thickness burns1.7.1.3 Deep partial-thickness burns; 1.7.1.4 Full-thickness burns; 1.7.1.5 Jackson zones and burn depth progression; 1.7.2 Types of burns; 1.7.2.1 Scald; 1.7.2.2 Grease; 1.7.2.3 Contact; 1.7.2.4 Flame; 1.7.2.5 Electrical contact; 1.7.2.6 Electrothermal (arc); 1.7.2.7 Chemical; 1.8 Animal models; 1.8.1 Injury types; 1.8.1.1 Granuloma models; 1.8.1.2 Incision models; 1.8.1.3 Open-wound models; 1.8.1.4 Burn models; 1.8.2 Impaired healing models; 1.8.2.1 Malnutrition; 1.8.2.2 Infection; 1.8.2.3 Ischemia; 1.9 Conclusion; Conflict of interest; References.
2.4.1 Expression of growth factors and their receptors2.4.2 Fibroblast proliferation; 2.4.3 Fibroblast migration; 2.4.4 Extracellular matrix synthesis and remodeling; 2.4.5 Contraction; 2.5 Growth factors, senescence, and wound healing; 2.6 Conclusion; References; 3 -- Targeting the myofibroblast to improve wound healing; 3.1 Introduction; 3.1.1 The stages of the wound healing process: timing is everything; 3.1.2 Inflammatory cells prepare the stage for fibroblasts; 3.2 From normal to abnormal wound healing: the myofibroblast; 3.2.1 Definition of the myofibroblast; 3.2.2 Myofibroblast markers.
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Item type Current library Call number Status Date due Barcode
Ebooks Ebooks Mysore University Main Library
Not for loan EBKELV270

Includes index.

Online resource; title from PDF title page (ScienceDirect, viewed June 21, 2016).

Front Cover; Wound Healing Biomaterials -- Volume 1; Related titles; Wound Healing Biomaterials: Volume 1: Therapies and Regeneration; Copyright; Contents; List of contributors; Woodhead Publishing Series in Biomaterials; One -- Fundamentals and strategies for wound healing; 1 -- Wound healing; 1.1 Introduction; 1.2 Skin layers; 1.3 Phases of wound healing; 1.3.1 Hemostasis and inflammation; 1.3.2 Proliferation; 1.3.3 Maturation and remodeling; 1.3.4 Wound contraction; 1.4 Growth factors and wound healing; 1.5 Acute and chronic wounds; 1.5.1 Acute wounds; 1.5.2 Chronic wounds.

1.5.2.1 Pressure ulcers1.5.2.2 Diabetic foot ulcers; 1.5.2.3 Arterial occlusive; 1.5.2.4 Venous stasis; 1.5.2.5 Lymphedema; 1.5.2.6 Calciphylaxis; 1.5.2.7 Warfarin-induced skin necrosis; 1.5.2.8 Infection; 1.6 Excessive scarring; 1.6.1 Hypertrophic scar; 1.6.2 Keloid; 1.6.3 Adhesions; 1.6.4 Pathophysiology; 1.6.5 Skin and scar evaluation; 1.6.6 Available treatment methods; 1.6.6.1 Intralesional injections; 1.6.6.2 Silicone gel; 1.6.6.3 Radiation; 1.6.6.4 Laser therapy; 1.6.6.5 Pressure therapy; 1.6.6.6 Surgery; 1.7 Burns; 1.7.1 Burn severity; 1.7.1.1 Superficial burns.

1.7.1.2 Superficial partial-thickness burns1.7.1.3 Deep partial-thickness burns; 1.7.1.4 Full-thickness burns; 1.7.1.5 Jackson zones and burn depth progression; 1.7.2 Types of burns; 1.7.2.1 Scald; 1.7.2.2 Grease; 1.7.2.3 Contact; 1.7.2.4 Flame; 1.7.2.5 Electrical contact; 1.7.2.6 Electrothermal (arc); 1.7.2.7 Chemical; 1.8 Animal models; 1.8.1 Injury types; 1.8.1.1 Granuloma models; 1.8.1.2 Incision models; 1.8.1.3 Open-wound models; 1.8.1.4 Burn models; 1.8.2 Impaired healing models; 1.8.2.1 Malnutrition; 1.8.2.2 Infection; 1.8.2.3 Ischemia; 1.9 Conclusion; Conflict of interest; References.

2.4.1 Expression of growth factors and their receptors2.4.2 Fibroblast proliferation; 2.4.3 Fibroblast migration; 2.4.4 Extracellular matrix synthesis and remodeling; 2.4.5 Contraction; 2.5 Growth factors, senescence, and wound healing; 2.6 Conclusion; References; 3 -- Targeting the myofibroblast to improve wound healing; 3.1 Introduction; 3.1.1 The stages of the wound healing process: timing is everything; 3.1.2 Inflammatory cells prepare the stage for fibroblasts; 3.2 From normal to abnormal wound healing: the myofibroblast; 3.2.1 Definition of the myofibroblast; 3.2.2 Myofibroblast markers.

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